PARKINSON'S DISEASE NEWS
28th July 2009 - New research
HIGH CARBOHYDRATE FOODS LESSEN PARKINSON'S DISEASE
Nutrition  Jul 21
[Epub ahead of print] (Murakami K, Miyake Y, Sasaki S, Tanaka K, Fukushima W,
Kiyohara C, Tsuboi Y, Yamada T, Oeda T, Miki T, Kawamura N, Sakae N, Fukuyama H,
Hirota Y, Nagai M; for the Fukuoka Kinki Parkinson's Disease Study Group.)
The researchers expected that foods such as these would decrease the risk of Parkinson's Disease by an insulin-induced increase in brain dopamine. Their theory appears to be correct, because high glycemic carbohydrates were inversely associated with the risk of Parkinson's Disease. The greater the intake, the less was the risk. No association was observed for other dietary carbohydrates, or dietary fiber intake. It was already known that an inability to make use of carbohydrates was common in Parkinson's Disease, with 50%-80% of people with Parkinson's Disease being prone to diabetes. In those people, higher carbohydrate intakes can not be made use of. For more information see the Complete abstract. In order to refer to this article on its own click here.
26th July 2009 - New report
AGENT ORANGE WRONGLY LINKED TO PARKINSON'S DISEASE
Veterans and Agent Orange Update
There have been over 300 published studies on the effects of Agent Orange, yet none of them have shown that Agent Orange has caused Parkinson's Disease. Claims of Agent Orange causing Parkinson's Disease have usually detailed how Parkinson's Disease was diagnosed years after possible exposure to Agent Orange. However, with Parkinson's Disease, if somebody is affected by a toxin, they usually suffer the effects at their worst soon after exposure to the toxin. So if Agent Orange caused the symptoms of Parkinson's Disease, they would have initiated whilst in Vietnam - not decades later. Somebody could be exposed to Agent Orange and quite independently develop Parkinson's Disease. Parkinson's Disease can occur in almost anyone without toxicity being the cause. In order to refer to this article on its own click here.
24th July 2009 - New book and DVD
MOVE IT, AN EXERCISE AND MOVEMENT GUIDE FOR PARKINSON'S DISEASE
22nd July 2009 - New research
ISTRADEFYLLINE FAILS CLINICAL TRIALS FOR PARKINSON'S DISEASE
Disorders  Jul 17 [Epub ahead of print] (Fernandez HH, Greeley DR, Zweig
RM, Wojcieszek J, Mori A, Sussman NM)
19th July 2009 - New research
THE EFFECT OF ADDING AGONISTS TO L-DOPA IN PARKINSON'S DISEASE
International Journal of Clinical Practice  63 (4) : 613-623 (Talati R,
Baker WL, Patel AA, Reinhart K, Coleman CI.)
Scores on the primary assessment of Parkinson's Disease, the UPDRS, are reduced when people added dopamine agonists to the existing use of L-dopa. They also experienced symptoms for less time, and were able to reduce their dosage of L-dopa. However, the incidence of dyskinesia and hallucinations was higher when dopamine agonists were added to the existing use of L-dopa. So the increase in efficacy was paid for with increased adverse events. Although the effect of L-dopa wears off in time, so does the effect of dopamine agonists. They work by stimulating the dopamine receptors. However, continuous use of dopamine agonists makes the dopamine receptors progressively less sensitive to dopamine and dopamine agonists. In order to refer to this article on its own click here.
17th July 2009 - New research
STEM CELLS MIGRATE IN PARKINSON'S DISEASE
 Jul 8. [Epub ahead of print] (Feng ZH, Ji MA, Li YU, Gang YU.)
The researchers claimed that this improved Parkinson's Disease. However, the Parkinson's Disease symptoms were only induced, and their methods did not actually assess improvements in Parkinson's Disease. Despite stem cell operations now being carried out around the world, they have never resulted in anyone being rid of Parkinson's Disease. Although it is claimed that stem cell operations are necessary because there is massive cell loss in Parkinson's Disease, no studies have ever shown that there is massive cell loss in Parkinson's Disease. In order to refer to this article on its own click here.
14th July 2009 - New research
GENES MULTIPLY THE EFFECT OF PESTICIDES ON PARKINSON'S DISEASE
perspectives  117 (6) : 964-969 (Ritz BR, Manthripragada AD, Costello S,
Lincoln SJ, Farrer MJ, Cockburn M, Bronstein J.)
The researchers do not explain how this defect can increase Parkinson's Disease. However, ridding dopamine too readily would explain the increased prevalence of Parkinson's Disease. In combination with exposure to pesticides, the risk of Parkinson's Disease was multiplied. Exposure to the pesticides paraquat and maneb, which are known causes of Parkinson's Disease, were increased by three times in those people that had one defect in the dopamine transporter, and by more than four times in those people that had two defects in the dopamine transporter. In some people the risk was many times greater than this. In order to refer to this article on its own click here.
12th July 2009 - New research
SCOLIOSIS IS PREVALENT IN PARKINSON'S DISEASE
Journal of Clinical
Neurology  5 (2) : 91-94 (Baik JS, Kim JY, Park JH, Han SW, Park JH, Lee
10th July 2009 - New research
GULF WAR VETERANS WITH PARKINSON'S DISEASE
American journal of
industrial medicine  Jul 7 [Epub ahead of print] (Barth SK, Kang HK,
Bullman TA, Wallin MT.)
However, Gulf War veterans potentially exposed to nerve agents at Khamisiyah, Iraq, and to oil well fire smoke had an increased risk of mortality due to brain cancer. As Parkinson's Disease is not a fatal illness, a better measure of the effects of the Gulf War on Parkinson's Disease would have been the prevalence of Parkinson's Disease in Gulf War veterans. However, no other studies have so far shown an increased prevalence of Parkinson's Disease in Gulf War veterans either. In order to refer to this article on its own click here.
9th July 2009 - News reports
INTERFERING WITH GLUTAMATE TO PREVENT PARKINSON'S DISEASE
It has been widely reported that researchers are aiming to interfere with the formation of Glutamate in order to prevent Parkinson's Disease. For the news reports go to Medical News Today and Science Daily. The research was recently presented at a conference. Glutamate is able to form GABA in the brain. GABA is a chemical produced naturally by the brain, that affects muscular function. An excess of GABA could provoke symptoms of Parkinson's Disease. The researchers aim to stimulate "trigger points" in order to prevent the release of glutamate. By targeting specific receptors they hope that side-effects will be minimised as fewer targets elsewhere in the brain will be stimulated.
They claim that glutamate causes cell death in Parkinson's Disease. However, glutamate formation is a healthy function, and has never been shown, in normal quantities, to cause cell death in people with Parkinson's Disease. The fundamental weakness in their theory is that glutamate has never been responsible for causing Parkinson's Disease when dopamine formation is sufficient either. The primary biochemical fault in Parkinson's Disease has been proven to be the insufficient formation of dopamine rather than an excess of glutamate. Yet the approach used by the researchers could not, even in theory, increase dopamine formation. In order to refer to this article on its own click here.
3rd July 2009 - New research
THE LACK OF CENTENARIANS WITH PARKINSON'S DISEASE
Journal of Rural Health
 Summer; 25 (3) : 320-325 (Kaye J, Michael Y, Calvert J, Leahy M, Crawford
D, Kramer P.)