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PARKINSON'S DISEASE NEWS |
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Parkinson's Disease News covers all significant new research, reports, books, and resources concerning Parkinson's Disease. Articles are chosen on the basis of their medical significance or potential interest. Our overwhelming priority is the facts, regardless of whether they contradict prevailing views or vested interests. Analysis and further information are provided either to explain the background or implications, or to balance misleading claims. If you notice errors or inadequacies, or dispute what is written, or want to propose articles, please e-mail [email protected].
19th June 2013 - New research SWEETENER FOR TREATING PARKINSON'S DISEASE Journal of Biological Chemistry [2013] 288 (24) : 17579-17588 (R.Shaltiel-Karyo, M.Frenkel-Pinter, E.Rockenstein, C.Patrick, M.Levy-Sakin, A.Schiller, N.Egoz-Matia, E.Masliah, D.Segal, E.Gazit) Complete abstract The sweetener, mannitol, has been proposed as a potential means of treating Parkinson's Disease because of the dual mechanisms it has in the brain. Mannitol, which is used in medicine, is derived from mannose, which is a sugar. For more information go to Mannitol.
The researchers therefore suggest mannitol as a basis for a dual mechanism therapeutic agent for the treatment of Parkinson's Disease. However, the research was only carried out on mice and flies, who did not have Parkinson's Disease and who were not rid of its symptoms. For a printable version of this article click here. For more news go to Parkinson's Disease News.
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Researchers
assessed the ability of mannitol to (1) interfere with the aggregation of
alpha-synuclein, and (2) its ability to disrupt the blood-brain barrier.
Alpha-synuclein can accumulate in the brains of people with Parkinson's Disease
and other medical disorders but can also be absent in Parkinson's Disease. It
therefore appears that Parkinson's Disease can cause alpha-synuclein rather than
alpha-synuclein being the cause of Parkinson's Disease as is often claimed. The
blood brain barrier restricts access to the brain to certain substances. They
demonstrated the effect of mannitol on alpha-synuclein by various means, and a
decrease in alpha-synuclein accumulation.
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